Have a lot more to add to the thread, back ground information, about the slew of diseases and conditions that are now being attributed (and a link in public mind forever sealed) to so many "deaths' this year from the super virus 'sars 2.0' or 'covid-19'.
First, a little background of our 'health care hero' Dr. Mike Hansen, his home page (link in his youtube videos),
https://doctormikehansen.com/
https://doctormikehansen.com/about/
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About Dr. Mike Hansen
Who Is Dr. Mike Hansen?
Mike Hansen, MD, is a medical doctor who is both an internist and an intensivist, as well as a pulmonologist. In other words, he specializes in (and is board certified in) internal medicine, pulmonary disease, and critical care medicine.
Between medical school, postgraduate training, and his actual practice of medicine, Dr. Hansen has dedicated over 15 years of his life to understanding how to prevent, diagnose, and treat diseases that affect adults.
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Perhaps not that unusual for a doctor to be an internist at other hospitals (around the country per se?) even after being in practice for at maybe 15 years? or maybe only 12 according to another link I saw but not including that one right now;
Michael Christopher Hansen MD
Pulmonology Valparaiso, IN
Critical Care Medicine, Pleural Disease
Physician
https://www.doximity.com/pub/michael-hansen-md-a48ae29e
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- New York Hospital Medical Center of QueensFellowship
- Lehigh Valley HospitalResidency
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St. George's University School of MedicineMedicalSchool
Certifications & Licensure
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FL State Medical License 2016 - 2018
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IN State Medical License 2015 - 2021
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NY State Medical License 2012 - 2019
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SD State Medical License Active through 2021
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Kinda weird why his picture is blurred out in this link. Need to register an account with Doximity to view his full profile, but you can't unless you are a,
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- Viewing the full profile is available to verified healthcare professionals only. Join over one million U.S. Physicians, Nurse Practitioners and PAs, already on Doximity.
- Find your profile and take control of your online presence:
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Dr. Michael Hansen, MD is a critical care medicine specialist in Valparaiso, IN and has over 15 years of experience in the medical field. He graduated from St. Georges University School of Medicine in 2008. He is affiliated with Monument Health Rapid City Hospital. His office accepts new patients.
www.healthgrades.com
No picture of Dr. Hansen in this link either. No reviews of Dr. Hansen on any link for him that I have seen in my googling so far. Or Duck Duck Go. I think we all know that their are probably automated bots that censor everybody's individual searches through generic or even specific search term flags and so forth. Makes it much harder for us to find smoking gun information as it were these days.
at end of his interview, teamed up with QB Tom Brady (New England Patriots)
https://doctormikehansen.com/interview/
about 44 videos in his library on this page (oldest one I think from Oct 2018 ), 28 I think are strictly about 'coronavirus/covid-19'.
https://doctormikehansen.com/videos/
No where on his site do I see where he practices medicine at on his personal site, but for now I'm going with that the Doximity and Healthgrade profile is the same doctor same guy. There is a contact form you can fill out, generates an e-mail to submit I suppose- that's what the "contact us" little boxes usually do.
https://doctormikehansen.com/contact/
from nrmis (post #43)
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Wow! What does influenza look like? Serious question.
Is there a test for influenza? Would influenza turn a cv19 pcr test to positive? Are any of these patients who look like they've got influenza being tested for influenza?
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You could try asking Dr. Mike these questions on his yourtube videos, seems he responds to some (probably very select) comments in the comment section of the videos.
The second video that Ruby posted from our 'health care hero',
12 Autopsy Cases Reveal TRUTH About How Patients Die From Coronavirus | COVID-19
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In all 12 cases, the cause of death was found within the lungs or the pulmonary vascular system. For the ones who did not die of large pulmonary emboli, they died of the extensive inflammation within the lungs, meaning pneumonia with ARDS. In these cases, the lungs were wet and heavy, much like a sponge that is saturated with water. The surfaces of the lung often had a distinct patchy pattern, with pale areas alternating with slightly protruding and firm, deep reddish-blue hypercapillarized areas. This is indicative of areas of intense inflammation, with endothelial dysfunction that can be seen at the microscopic level. When they look at slices of the lungs under the microscope, they found diffuse alveolar damage in 8 cases. Specifically, they saw hyaline membrane formation, and tiny clots in the capillaries, and capillaries that were engorged with red blood cells, and other inflammatory findings. All these findings represent ARDS. They also found lymphocytes, a type of white blood cell, infiltrated these areas of infiltration. This fits the picture of viral pathogenesis.
They also looked at the pharynx of these patients, meaning in their throat. The lining of the throat, or mucosa, was hyperemic, meaning very red and irritated, and at the microscopic level, they saw lymphocytes invading there, which is consistent with a viral infection. In one case, a patient had lymphocytes invade his heart muscle, findings that are consistent with what we call viral myocarditis. More than half of the patients in this study had large blood clots. One-third of the patients had pulmonary embolism as the direct cause of death. All the others died of intense inflammation in their lungs related to pneumonia with ARDS (Acute Respiratory Distress Syndrome). Recently there’s been studies showing that about 1/3rd of patients with severe COVID have blood clots. Another study of 191 patients with coronavirus aka COVID-19, half of those who died had clots, compared with 7% of survivors. And levels of D-dimer that were greater than 1000 µg/L were associated with a fatal outcome. So it's pretty clear now that the SARS-CoV-2 virus is causing a lot of clots to form in moderate to severe COVID disease.
How is this happening? It's likely a combination of reasons, that has to do with downregulation of the ACE2 receptor in the lung alveoli, with a subsequent shift towards having more angiotensin II in the lungs, and less angiotensin 1-7 and 1-9 in the lungs, and when this happens, this leads to more cytokine storm with more inflammation, more constriction of pulmonary arteries, and more clots that develop. That, in turn, leads to more endothelial dysfunction in the capillaries that surround the alveoli. Also, there is evidence that the virus attaches to the ACE2 receptors of those endothelial cells that line those capillaries, which further propagates inflammation and clotting. And in the cytokine storm that develops there, RANTES, a chemokine, binds to the CCR5 receptor of CD4 and CD8 lymphocytes, and that causes those lymphocytes to infiltrate those areas of inflammation, and in doing so, further contributes towards the inflammatory reaction. This is why we are seeing low levels of CD4 and CD8 lymphocytes in severe COVID. Endothelial damage can also lead to the development of antiphospholipid antibodies, and these antibodies are bad because they trigger the formation of blood clots. That’s why patients who have clots with the diagnosis of antiphospholipid antibody syndrome need to be on blood thinners.
Also, 11 out of the 12 patients in this study had underlying heart disease and were obese. These are known risk factors not just for cardiovascular disease, but also known risk factors for endothelial dysfunction, and are known risk factors for COVID. So the big takeaways from the findings in this study are that most people who die of COVID, it's primarily a lung problem. Either related to inflammation with ARDS and/or blood clots.
Antiphospholipid syndrome might be a commonality among patients with thrombosis in COVID-19 patients. Dr. Mike Hansen, MD Internal Medicine | Pulmonary Disease | Critical Care Medicine Website:
https://doctormikehansen.com/
Instagram Account: http://instagram.com/doctor.hansen/
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I bolded near the last paragraph of the video description/transcript (sort of maybe), I think it relevant to point out these (and plenty of other "risk factors" in a slew of diseases I will bring up later too) are known risk factors for a whole lot of things, and tons of risk factors and pre-existing conditions in the vast majority of people whom have alleged to have died from 'covid-19'.
In order to give a least a minimal breakdown of what he presents, and all the other conditions/diseases that I'm taking about, it will take probably quite a few posts to fill in enough information and to show (at least a strong circumstantial case), or demonstrate as simply as possible, links, correlations, some videos, articles, diagrams etc., to support my assertion that 'covid-19' is a cover for a very wide variety of diseases, in order to promote (as well as many 'deaths' as attributed to as possible) this fake and planned 'pandemic' for obviously a much larger agenda(s).
That is not going to be easy at all. For now, I will add just a two topics that go along with filling in little bit of background info in pursuit of showing or proving (again not absolute proof) that 'covid-19' is being used as a cover (up) for other diseases to promote the end game agenda.
Pulmonary Fibrosis
Pulmonary Embolism (and related, deep vein thrombosis or DVT)
Pulmonary Fibrosis Foundation
https://www.pulmonaryfibrosis.org/
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COVID-19 Operational Impact
Please note that a statewide "stay-at-home" order has been issued by the governor of Illinois until May 30. The office building in which the PFF is located has been closed during
this order. PFF staff are working remotely and will not be able to process requests for hard-copies of educational materials and will also not be able to process donations mailed to our office
until the building re-opens. All online donations will continue to be acknowledged in a timely manner. We thank you for your patience during this time.
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One little notice that goes along with the notion that some people who have existing illness, and ones that are potentially fatal without hospital medical treatment in some cases, may not be getting the care they need in last three months and succumbed to that disease, and many have probably still ended up as a 'covid' death statistic despite having died at home and likely no 'covid' 'test' performed. Well let me correct this comment, this is just the notice about the
Pulmonary Fibrosis Foundation office being closed, not necessarily medical offices, for PF or other any conditions but that is still probably likely the case, but this site is not direct proof of that.
Proof of that would have been something very helpful that firetur.., er I mean firetown could have (and should have) provided but didn't, to show that this is the case, people dying at home due to medical services being cut off. I will try at some point to show at least one source of "evidence" for that in this thread.
PF overview from the Pulmonary Fibrosis Foundation,
https://www.pulmonaryfibrosis.org/life-with-pf/about-pf
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Pulmonary Fibrosis Overview
Feeling confused or overwhelmed about pulmonary fibrosis or idiopathic pulmonary fibrosis? You are not alone. The Pulmonary Fibrosis Foundation is here to help you understand what it means to have pulmonary fibrosis. You can always reach us through our Patient Communication Center at 844-825-5733 or by email at pcc@pulmonaryfibrosis.org.
Please remember that this information is a brief overview and is for educational purposes only. It is not intended to be a substitute for professional medical advice. Always consult your health care provider with any questions you may have regarding your specific medical condition.
To find a local pulmonary fibrosis support group near you, click here.
What is Pulmonary Fibrosis?
The word “pulmonary” means lung and the word “fibrosis” means scar tissue— similar to scars that you may have on your skin from an old injury or surgery. So, in its simplest sense, pulmonary fibrosis (PF) means scarring in the lungs. Over time, the scar tissue can destroy the normal lung and make it hard for oxygen to get into your blood. Low oxygen levels (and the stiff scar tissue itself) can cause you to feel short of breath, particularly when walking and exercising. Pulmonary fibrosis isn’t just one disease. It is a family of more than 200 different lung diseases that all look very much alike. The PF family of lung diseases falls into an even larger group of diseases called the interstitial lung diseases (also known as ILD), which includes all of the diseases that have inflammation and/or scarring in the lung. Some interstitial lung diseases don’t include scar tissue. When an interstitial lung disease does include scar tissue in the lung, we call it pulmonary fibrosis.
No one is certain how many people are affected by PF. One recent study estimated that idiopathic pulmonary fibrosis (or IPF, which is just one of more than 200 types of PF) affects 1 out of 200 adults over the age of 60 in the United States—that translates to more than 200,000 people living with PF today. Approximately 50,000 new cases are diagnosed each year and as many as 40,000 Americans die from IPF each year.
Find expert care for pulmonary fibrosis.
www.pulmonaryfibrosis.org
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Break, more from this section,
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Pulmonary Fibrosis Symptoms and Causes
It can be challenging for doctors to figure out what causes PF. Sometimes they are able to identify one or more causes of your disease, which are discussed here. PF of unknown cause is called “idiopathic”
There are five main categories of identifiable causes of pulmonary fibrosis: Drug-induced, Radiation-induced,
Environmental,
Autoimmune, and Occupational. In the United States, Environmental and Autoimmune causes seem to be the most common types of PF of known cause.
This table shows some of the clues that doctors use to identify these known causes of PF.
TYPE OF PULMONARY FIBROSIS CLUES THAT DOCTORS USE
Drug-induced Prior or current use of amiodarone, nitrofurantoin, chemotherapy, methotrexate, or other drugs known to affect the lungs Radiation-induced Prior or current radiation treatment to the chest Environmental (called hypersensitivity pneumonitis) Exposure to mold, animals, or other triggers ("squeaks" heard in the lung) Autoimmune (called connective tissue disease-related) Joint inflammation, skin changes (particularly on the fingers and face), dry eyes or mouth, abnormal blood tests Occupational (called pneumoconiosis) Prior or current exposure to dusts, fibers, fumes, or vapors that can cause PF (such as asbestos, coal, silica, and others) Idiopathic When no cause can be identified
Medications That Cause Pulmonary Fibrosis
Some
MEDICATIONS can cause PF. Drugs used to treat cancer (chemotherapy), drugs used to treat abnormal heart rhythms (such as amiodarone), drugs used to treat inflammatory conditions (such as methotrexate), and an antibiotic used to treat urinary tract infections (nitrofurantoin) are some of the better known drugs that can cause injury, inflammation, and scarring in the lungs. Numerous other drugs have been implicated as causes of PF in some cases.
Pulmonary Fibrosis Caused by Radiation
RADIATION to the chest for lymphoma; Hodgkin’s disease; or breast, lung, and other cancers can injure the lung and cause fibrosis.
Environmental Causes of PF
are typically called hypersensitivity pneumonitis (HP) or chronic hypersensitivity pneumonitis. HP occurs when the lungs react with inflammation and scarring after breathing in mold spores, bacteria, animal proteins (especially from indoor or caged birds), or other known triggers. No one is certain why some people are so susceptible to developing HP and others are not.
Pulmonary Fibrosis and Autoimmune Diseases
AUTOIMMUNE DISEASES are also called connective tissue diseases, collagen vascular diseases, or rheumatologic diseases. “Auto” means self and “immune” refers to your immune system. So if you have an autoimmune disease affecting your lungs, it means that your body’s immune system is attacking your lungs. Examples of autoimmune diseases that can cause PF include
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- Rheumatoid arthritis;
- Scleroderma (also called systemic sclerosis);
- Sjögren’s syndrome; and
- Polymyositis, dermatomyositis, and antisynthetase syndrome.
Occupational Causes of PF
Occupational causes also called pneumoconiosis, can develop after significant exposure to a wide variety of inorganic dusts, including asbestos, silica, coal dust, beryllium, and hard metal dusts.
Is Pulmonary Fibrosis Hereditary?
Yes, but in most cases, it is not as straightforward as inheriting blue eyes or red hair. There are three different scenarios:
1) Did the genes I inherited from my parents contribute to PF even if I am the only one in my family with PF?
Yes. The risk of developing most human diseases is influenced by the genes you inherited from your parents. There have been several genes identified that appear to increase the risk of developing PF when an abnormal form of the gene is inherited. There are no official medical guidelines on whether or not genetic testing should be performed if you have PF, although in some cases, your doctor might wish to test you for one or more abnormal forms of these genes.
2) Can PF run in the family?
Yes. Anywhere from three to 20% of people with PF have another family member with PF. But this doesn’t mean that up to 20% of your family members will develop PF. In most cases, the chance that one of your family members will develop PF is very low. We encourage you to talk to your doctor or to a genetic counselor about the chances that one of your family members will develop PF.
3) Are there specific types of PF where the entire disease is due to one gene?
Yes. There are a few rare genetic forms of PF that can affect both children and adults when just a single gene is inherited in an abnormal form (usually from both parents). Hermansky-Pudlak syndrome is an example of a genetic form of PF that simply requires abnormalities in one gene. People living with Hermansky-Pudlak syndrome often also have very light skin pigmentation (due to low levels of melanin in their skin) and bleeding problems (due to poorly functioning platelets in their blood). Dyskeratosis congenita is another example of a genetic form of PF that can be accompanied by poorly growing fingernails, changes in skin pigmentation, increased risks of developing problems in the bone marrow, and other conditions.
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Treatment Options
The Pulmonary Fibrosis Foundation also provides details on the latest
treatment options and
drug development breakthroughs to help patients live a stronger life. If you are interested in learning about another form of PF, Idiopathic Pulmonary Fibrosis, visit our dedicated
Idiopathic Pulmonary Fibrosis page. Treatment options available as well.
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What Is Idiopathic Pulmonary Fibrosis?
Idiopathic Pulmonary Fibrosis (IPF) is a scarring disease of the lungs of unknown cause. To make a diagnosis of IPF, your doctor will perform a thorough history to try to identify potential exposures or other diseases that might lead to scarring of the lung.
If diagnosed, over time scarring can worsen making it hard to take a deep breath. The lungs then cannot take in enough oxygen to oxygenate the blood. IPF is a form of interstitial lung disease, primarily involving the interstitium or the tissue and space around the air sacs of the lungs, and not directly affecting the airways or blood vessels.
There are various kinds of interstitial lung disease that can also be caused by inflammation and/or fibrosis. These types of IPF are treated a bit differently. It is important to work with your health care provider to determine if you have IPF or another form of interstitial lung disease. If a plausible cause is found, then you do not have IPF. The scarring pattern of IPF is technically called usual interstitial pneumonia (UIP). Your doctor will use detailed X-rays of your lungs called high-resolution computed tomography (HRCT) and sometimes a lung biopsy to look for this pattern. A diagnosis of IPF requires that your doctor cannot find a cause and the presence of a pattern of UIP on either HRCT or a surgical lung biopsy sample.
Although IPF is still considered to be a disease of unknown cause, we do know some factors that increase the risk of getting IPF, including aging (IPF is rare before age 50), cigarette smoking, and having certain genetic predispositions.
Bolded relevant symptom here, sound familiar? like 'covid'?
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Signs & Symptoms of Idiopathic Pulmonary Fibrosis
The most common symptoms of IPF are dry, hacking cough and shortness of breath. Symptoms may be mild or even absent early in the disease process. As the lungs develop more scar tissue, symptoms worsen. Shortness of breath initially occurs with exercise, but as the disease progresses patients may become breathless while taking part in everyday activities, such as showering, getting dressed, speaking on the phone, or even eating.
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supposedly now some people alledgedly having 'covid' symptoms for months? PF and IPF can last for years and is quite often fatal.
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Due to a lack of oxygen in the blood, some people with idiopathic pulmonary fibrosis may also have “clubbing” of the fingertips. Clubbing is a thickening of the flesh under the fingernails, causing the nails to curve downward. It is not specific to IPF and occurs in other diseases of the lungs, heart, and liver, and can also be present at birth.
Other common symptoms of pulmonary fibrosis include:
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- Fatigue and weakness
- Discomfort in the chest
- Loss of appetite
- Unexplained weight loss
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Pulmonary Fibrosis Diagnosis
How do doctors recognize and diagnose pulmonary fibrosis? There are three consequences of PF. Doctors use these consequences to recognize that someone has PF.
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- STIFF LUNGS
— Scar tissue and inflammation make your lungs stiff. Stiff lungs are hard to stretch, so your breathing muscles have to work extra hard just to pull air in with each breath. Your brain senses this extra work, and it lets you know there’s a problem by triggering a feeling of breathlessness(or “shortness” of breath) while exerting yourself. Also, stiff lungs hold less air (they shrink up a bit). Doctors take advantage of this “shrinking” to diagnose and track the disease using breathing tests (called Pulmonary Function Tests) that measure how much air your lungs can hold. The more scar tissue your lungs have, the less air they will hold.
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- LOW BLOOD OXYGEN
— Scar tissue blocks the movement of oxygen from the inside of your air sacs into your bloodstream. For many people living with pulmonary fibrosis, oxygen levels are only reduced a little bit while resting, but their oxygen levels drop quite a bit during exertion. The brain can sense these low oxygen levels, triggering breathlessness. Doctors will check your oxygen levels to see if they drop after walking, a clue that PF might be present. Doctors also often prescribe oxygen to be used through a nasal cannula or a facemask during exertion and sleep for those with PF. As pulmonary fibrosis progresses, oxygen may be needed 24 hours a day.
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- CRACKLES LUNG SOUNDS
— Your doctor may have told you that “crackles” were heard in your lungs. Crackles (also called “rales”) sound like Velcro being pulled apart. They are heard in many lung diseases because any type of problem affecting the air sacs (such as PF, pneumonia, or a buildup of fluid in the lungs from heart failure) can cause crackles. Some people with pulmonary fibrosis don’t have crackles, but most do.
Once your doctor recognizes that you might have pulmonary fibrosis, the next step is to try to diagnose the specific kind of PF you have— there are more than 200 different kinds. Doctors typically start by asking many questions, performing a careful physical examination, and ordering a lot of blood tests. See Causes for more information.
A doctor will also use a special kind of X-ray of the chest, called a high-resolution computed tomography (HRCT) scan, so that they can see what your lung tissue looks like. HRCT scans give a close-up view of the lungs, providing more detail than routine CT scans (also known as CAT scans). Healthy lung tissue looks nearly black on a CT scan. Scar tissue and inflammation both appear grey or white. Many forms of PF look similar on a CT scan to the untrained eye, but subtle findings on HRCT scans are critically important when trying to identify which type of PF you have.
Sometimes, even after all of the testing is complete, a doctor will still not have an answer and will have to perform a lung biopsy to sort out which of the 200 different types of pulmonary fibrosis you have. When indicated, a lung biopsy can also help you and your doctor decide which treatments might be helpful.
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Newly Diagnosed with Pulmonary Fibrosis?
I was just diagnosed with pulmonary fibrosis. What do I do next?
We suggest that you consider making an appointment with a pulmonologist who has experience caring for patients with PF. A knowledgeable team of PF experts will help make sure you receive an accurate diagnosis and the most up-to-date treatments and management recommendations. To assist you in identifying pulmonologists closer to home and developing expertise in the care of patients with PF, the Pulmonary Fibrosis Foundation established the PFF Care Center Network that includes 60 medical centers throughout the United States. You can find a list of sites within the Network at
pulmonaryfibrosis.org/life-with-pf/find-medical-care.
We also recommend that you consider joining a pulmonary fibrosis support group. Connecting with other individuals facing the same illness can help you and your family not feel so alone in your journey with pulmonary fibrosis. Support groups can supplement the care you receive from your health care team by providing emotional support and education.
Support groups can help those living with pulmonary fibrosis
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- learn about their disease and available treatments;
- feel supported by others who are going through the same thing;
- learn to navigate the health care system more effectively; and
- improve coping skills, among other things.
© 2019 Pulmonary Fibrosis Foundation
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Another PF foundation
IPF Foundation
https://ipffoundation.org/
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- A bacterial protein fragment kills lung cells in pulmonary fibrosis. Learn more.1, 2
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- 31% of first degree relatives of patients with pulmonary fibrosis had interstitial lung abnormalities on CT. Learn more.3
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- The incidence of idiopathic pulmonary fibrosis (IPF) doubled over the decade, according to the New England Journal of Medicine. Learn more.4
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- The CDC identified a statistically significant cluster of IPF patients diagnosed at a Virginia medical center. Learn more.5
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- An estimated 50,000 people die from IPF in the U.S. each year, more deaths than from breast cancer. (See source study.) This statistic does not include the high percentage of people who are never diagnosed. See graphic below. Experts agree that we don’t know the extent of deaths and that more research is needed.6
(One of the references from above)
4. Verma, Subodh, Slutsky, Arthur S. “Idiopathic Pulmonary Fibrosis — New Insights.” New England Journal of Medicine, vol. 356, no. 13, 2007, pp.
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I will have to come back and add more, break down some stuff to fill in the framework of the basic hypothesis etc., gotta cut this off for now.
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